Thyroid assessment.pdf

THYROID ASSESSMENT
Hair tissue mineral analysis offers excellent information about thyroid activity and often very different information than blood tests. The hair assessment can be extremely helpful, especially in a common syndrome that one might call secondary hyperthyroidism. Most confusion arises because blood thyroid tests do not reveal much about thyroid physiology. Standard tests only measure circulating hormones (T3 and T4) and pituitary stimulation of the thyroid (TSH). In most cases, thyroid problems can be corrected without using natural or synthetic hormone replacement. When needed, natural thyroid supplementation is usually far superior to
synthetic drugs such as Synthroid or Levoxyl. Thyroid-inhibiting drugs, RAI (radioactive
iodine) or thyroid surgery are never required in my experience. In many cases, these treatments
are absolutely incorrect once one understands the physiology.
THYROID PHYSIOLOGY
Thyroid metabolism involves four important stages:
1) Hormone Production. To produce thyroxine (T4) requires manganese, iodine, tyrosine,
cyclic AMP, vitamin C and B-complex, and other micronutrients. Radiation toxicity, excessive
oxidant stress or toxic chemicals can block hormone synthesis. Mercury and copper toxicity
stimulate hormone synthesis.
2) Hormone Release. Secretion of thyroid hormones requires sympathetic nervous stimulation.
Many people have exhausted adrenals or other autonomic imbalances that may affect the
sympathetic nervous system.
3) Absorption into the Cells. Once released into the blood, T4 must be absorbed into the body
cells. For this to occur, the cell membranes must function properly. Accumulation of
biounavailable calcium and magnesium excessively stabilize cell membranes and reduce cell
permeability. Deficient calcium and magnesium cause excessive cell permeability. Oxidant stress
or impaired fatty acid metabolism or other damage to cell membranes can also block absorption
of thyroxine.
Copper affects absorption by altering calcium and potassium levels. Cadmium or nickel toxicity affect hormone absorption by affecting the levels of calcium, sodium and other critical
minerals.
4) Utilization in the Mitochondria. Once inside the cells, thyroxine must be converted to to T3
and utilized in the mitochondria. Potassium plays a role in sensitizing the mitochondria to
thyroid hormone. Fluorides in drinking water and chlorides found in bleaches used to make
white flour are powerful inhibitors of thyroid hormone utilization. They interfere with iodine
metabolism. Substances in soy and in raw cabbage, cauliflower and broccoli also inhibit thyroid
hormone utilization. The worst foods for one's thyroid are soy products and foods made with
white flour. Many packaged foods processed with water contain high levels of fluorides that
have found their way into water supplies.
Cells must also be able to respond to thyroid hormone stimulation. A range of vitamins and minerals are required for energy production in the glycolysis and carboxylic acid cycles in
the mitochondria. If these co-factors are missing or toxins block steps in the pathway, thyroid
hormone will be ineffective in increasing energy production.
THYROID PROBLEMS
Imbalances can occur at any stage of the production or utilization of thyroid hormone. The concepts of hypothyroidism and hyperthyroidism are incomplete and often misleading as they only relate to hormone production and release. One person might have inadequate hormone production due to radiation damage. Another produces enough hormone, but has an autonomic imbalance preventing its release. Another cannot transport enough hormones into the cells due to low cell permeability. Still another person might have adequate hormone production but be unable to utilize the hormones in the cells due to manganese deficiency or fluoride toxicity. Another may have excess hormone production due to copper or mercury toxicity and at the same time have inadequate cell permeability, causing a mixture of hypo- and hyperthyroid symptoms. Blood tests do not assess these factors. As a result, they miss many problems, may indicate a problem where none exists or may indicate one imbalance when the opposite condition exists at the cellular level. Most commonly, serum thyroid tests are normal but a thyroid imbalance is present. This may occur because the normal ranges of the blood tests are too large. TSH should not be above 3.5, yet many doctors still use 5 as the upper limit of normal. In other cases, however, the blood tests cannot detect deficiencies and toxins affecting thyroid activity. The most common imbalances are low thyroid effect due to impaired cell permeability in slow metabolizers and hyperthyroid symptoms due to copper or mercury toxicity of the thyroid
gland. These commonly occur together causing a mixture of symptoms.
HAIR ANALYSIS FOR THYROID ASSESSMENT
Hair analysis is excellent to help assess thyroid difficulties. It can indicate imbalances in many steps involved in thyroid hormone metabolism:
PRIMARY HYPERTHYROIDISM

In these cases, which are less common than secondary hyperthyroidism, the hair mineral analysis will reveal a fast oxidation rate. That is, calcium is often less than 40 mg% or 400 ppm and magnesium less than 6 mg% or 60 ppm. Sodium and potassium are often high if the hair is not washed at the laboratory. The thyroid is overactive, usually due to the presence of toxic metals such as mercury in the thyroid or pituitary glands or other toxic metals such as copper, lead, iron, cadmium or others. Metabolic balancing is usually very effective at restoring normal thyroid activity. SECONDARY HYPERTHYROIDISM
This is the most common type of hyperthyroidism in my experience. Serum levels of T3 and T4 are often elevated. TSH may be high, normal or low. The patient will have symptoms of hyperthyroidism such as anxiety and exothalmos. Yet the patient is often fatigued as well. The hair analysis will show a slow oxidizer pattern, often with very low sodium and potassium levels and quite an elevated calcium level. In these cases, the cells are not permeable enough to thyroid hormone due to elevated calcium. Low potassium may impair the sensitivity of the tissues to thyroid hormone. In
response, the body may elevate T4 and/or T3 production as a compensation. Very often, the
patient pushes herself by doing too much or exercising excessively. This must stop for
correction to occur. Then metabolic correction often works well and rapidly to correct this
imbalance.
PRIMARY HYPOTHYROIDISM

A hair calcium above 50 mg% and magnesium above about 9 mg% indicate some degree of biounavailable calcium and magnesium. This occurs mainly in slow metabolizers. One is
often fatigued and may have other symptoms of low thyroid activity such as dry skin and hair,
weight gain (though in some cases one is very thin), cold intolerance and others. If serum
hormone levels are normal or elevated, physicians may not give thyroid support when it is in fact
needed.
WHEN THYROID HORMONE IS HARMFUL

When hair tissue calcium and magnesium are low, as in fast metabolizers, cell membranes are more permeable. This causes more rapid uptake of thyroid hormone into the cells
and an increased thyroid effect. The patient may present with fatigue or other symptoms
suggestive of low thyroid activity. Serum thyroid hormone levels may be low normal or even
decreased. A physician who only measures serum hormone levels (T3 and T4) or TSH might
conclude that the patient needs thyroid hormone. This will make the patient's condition much
worse, although it may provide a temporary energy boost. * The hair calcium level is an
approximate thyroid effect indicator because thyroid hormone lowers calcium in the body. The
higher the level of hair calcium, in general, the lower the effective activity of the thyroid gland.
OTHER HAIR THYROID INDICATORS

* The potassium level is associated with sensitivity of the tissues to thyroid hormone. Low hair
potassium is associated with reduced sensitivity of the mitochondrial receptors to thyroid
hormone. Even if circulating hormone levels are normal and hormones can be absorbed into the
cells, when tissue potassium is low they may not be utilized, resulting in a low thyroid effect.
This commonly contributes to thyroid problems in slow metabolizers. Potassium supplements
rarely help because the problem is a loss of potassium due to kidney dysfunction and electrical
imbalances at the cellular level.
* Manganese deficiency can reduce thyroid activity. Manganese is required for T4 production. Manganese deficiency or bio-unavailability are very common today. Deficiency is associated with a hair manganese level less than 0.03 mg%. A level greater than 0.07 mg% often indicates bio-unavailability. Adrenal exhaustion causes manganese to become bio-unavailable as the binding protein, transmanganin, is not produced in sufficient quantity. * Metabolic typing can assess vitamin needs. Vitamins C and B-complex, for instance, tend to enhance thyroid activity. Higher doses are given to slow metabolizers and less to fast metabolizers to help balance thyroid activity. Supplementation without testing for the metabolic type is often ineffective or can aggravate thyroid imbalances. * Hair calcium and magnesium levels are associated with cell permeability. Bio-unavailable calcium and magnesium stabilize cell membranes. This causes reduced cell membrane permeability that decreases thyroid hormone uptake into the cells. This produces a cellular thyroid hormone deficiency. Serum hormone levels may be normal or even elevated. When the serum levels are elevated and the hair test shows a high calcium and low potassium, it is a secondary hyperthyroidism. * Copper is an important thyroid indicator. The key here is that one cannot use the hair copper level as the only copper indicator because copper often does not accumulate in the hair, but rather in the brain, liver and other organs. One must not supplement copper simply on the basis of the hair copper level. Other test numbers, however, offer excellent information about copper status: 1) Copper raises calcium and lowers potassium. Elevated calcium and low potassium is a slow metabolizer pattern associated with the presence of excess tissue copper. It does not matter if the hair copper is low, normal or high. The pattern is associated with reduced thyroid utilization and hypothyroidism. 2) Compensatory effects may occur. Copper stimulates the production of biogenic amines - epinephrine, norepinephrine and dopamine. These can cause anxiety, sweating and other symptoms similar to hyperthyroidism. The body may compensate for the inhibitory effect of high calcium and low potassium by increasing T3 and T4 to force more thyroid hormone into the cells. TSH may vary. The symptoms and blood tests cause some physicians to diagnose hyperthyroidism. Irradiation or even surgery may be recommended when the real problem is copper imbalance. This occurs commonly. 3) Weak adrenals cause copper to become bio-unavailable. This produces another mixed picture. Often this is indicated by a sodium/potassium ratio less than 2:1 or a hair copper less than 1.0 mg%. In these cases, even if hair copper is high, one must give some copper to relieve symptoms until copper becomes biologically available. 4) Fast metabolizers are copper deficient. They have a relatively low hair calcium level and elevated hair potassium. Their cells are excessively permeable and sensitive to thyroid hormone. Fast metabolizers all require copper supplements even if their hair copper level is normal or elevated. * Other Toxic Metals and Imbalances. Energy production requires many nutrients, and can be blocked by toxic chemicals and heavy metals. Hair analysis may provide indicators of an impaired energy such as cadmium toxicity or zinc deficiency that causes thyroid hormone to be ineffective in stimulating energy production. * Autonomic Balance. Most slow metabolizers have depleted their sympathetic nervous systems and are in a pathological parasympathetic state. This can affect thyroid hormone release. The Eck Institute Bulletin Volume 18 Dec. 2002 Number 12 Home * Hair Analysis * Saunas * Books * Articles Detoxification Protocols * Courses * About Dr. Wilson

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